Education | Insight Maker

Despite a mature field of inquiry, frustrated educational policy makers face a crisis characterized by little to no clear research-based guidance and significant budget limitations -- in the face of too often marginal or unexpectedly deleterious achievement impacts. As such, education performance has been acknowledged as a complex system and a general call in the literature for causal models has been sounded. This modeling effort represents a strident first step in the development of an evidence-based causal hypothesis: an hypothesis that captures the widely acknowledged complex interactions and multitude of cited influencing factors. This non-piecemeal, causal, reflection of extant knowledge engages a neuro-cognitive definition of students. Through capture of complex dynamics, it enables comparison of different mixes of interventions to estimate net academic achievement impact for the lifetime of a single cohort of students. Results nominally capture counter-intuitive unintended consequences: consequences that too often render policy interventions effete. Results are indexed on Hattie Effect Sizes, but rely on research identified causal mechanisms for effect propagation. Note that the net causal interactions have been effectively captured in a very scoped and/or simplified format. Relative magnitudes of impact have been roughly adjusted to Hattie Ranking Standards (calibration): a non-causal evidence source. This is a demonstration model and seeks to exemplify content that would be engaged in a full or sufficient model development effort. Budget & time constraints required significant simplifying assumptions. These assumptions mitigate both the completeness & accuracy of the outputs. Features serve to symbolize & illustrate the value and benefits of causal modeling as a performance tool.

Clone of Version 10: Hattie Calibrated Education Scenario Tool Capability Demonstration

JMantilla

Pennthorpe Architecture

Neil Jones

WIP based on Raafat Zaini's 2015 Triple Helix article and PhD Colloquium and ISDC 2013 university growth paper ithink models as a starting point for health care systems science modelling growth dynamics

Clone of Startup University Model

Josh Hawley

This is a simple population model designed to illustrate some of the concepts of stock and flow diagrams and simulation modelling.

We replaced the variable "Life Expectancy" with a converter that allows us to indicate how life expectancy declines over time with an increasing population (due to scarcity of resources, urban crowding, disease, etc) assuming no further technological or medical advancements.

Population Stock and Flow with Declining Age Expectancy

Stephanie Krishnan

Physical meaning of the equations

The Lotka–Volterra model makes a number of assumptions about the environment and evolution of the predator and prey populations:

1. The prey population finds ample food at all times.

2. The food supply of the predator population depends entirely on the size of the prey population.

3. The rate of change of population is proportional to its size.

4. During the process, the environment does not change in favour of one species and genetic adaptation is inconsequential.

5. Predators have limitless appetite.

As differential equations are used, the solution is deterministic and continuous. This, in turn, implies that the generations of both the predator and prey are continually overlapping.[23]

Prey

When multiplied out, the prey equation becomes

dx/dt = αx - βxy

The prey are assumed to have an unlimited food supply, and to reproduce exponentially unless subject to predation; this exponential growth is represented in the equation above by the term αx. The rate of predation upon the prey is assumed to be proportional to the rate at which the predators and the prey meet; this is represented above by βxy. If either x or y is zero then there can be no predation.

With these two terms the equation above can be interpreted as: the change in the prey's numbers is given by its own growth minus the rate at which it is preyed upon.

Predators

The predator equation becomes

dy/dt = -

In this equation, {\displaystyle \displaystyle \delta xy} represents the growth of the predator population. (Note the similarity to the predation rate; however, a different constant is used as the rate at which the predator population grows is not necessarily equal to the rate at which it consumes the prey). {\displaystyle \displaystyle \gamma y} represents the loss rate of the predators due to either natural death or emigration; it leads to an exponential decay in the absence of prey.

Hence the equation expresses the change in the predator population as growth fueled by the food supply, minus natural death.

Clone of Prey&Predator

Jun UMEDA

11 months ago

Student Completing Homework System Thinking

Alexander Thanasoulis

Despite a mature field of inquiry, frustrated educational policy makers face a crisis characterized by little to no clear research-based guidance and significant budget limitations -- in the face of too often marginal or unexpectedly deleterious achievement impacts. As such, education performance has been acknowledged as a complex system and a general call in the literature for causal models has been sounded. This modeling effort represents a strident first step in the development of an evidence-based causal hypothesis: an hypothesis that captures the widely acknowledged complex interactions and multitude of cited influencing factors. This non-piecemeal, causal, reflection of extant knowledge engages a neuro-cognitive definition of students. Through capture of complex dynamics, it enables comparison of different mixes of interventions to estimate net academic achievement impact for the lifetime of a single cohort of students. Results nominally capture counter-intuitive unintended consequences: consequences that too often render policy interventions effete. Results are indexed on Hattie Effect Sizes, but rely on research identified causal mechanisms for effect propagation. Note that the net causal interactions have been effectively captured in a very scoped and/or simplified format. Relative magnitudes of impact have been roughly adjusted to Hattie Ranking Standards (calibration): a non-causal evidence source. This is a demonstration model and seeks to exemplify content that would be engaged in a full or sufficient model development effort. Budget & time constraints required significant simplifying assumptions. These assumptions mitigate both the completeness & accuracy of the outputs. Features serve to symbolize & illustrate the value and benefits of causal modeling as a performance tool.

Version 11: Hattie Calibrated Education Scenario Tool Capability Demonstration

Yolande Tra

Physical meaning of the equations

The Lotka–Volterra model makes a number of assumptions about the environment and evolution of the predator and prey populations:

1. The prey population finds ample food at all times.

2. The food supply of the predator population depends entirely on the size of the prey population.

3. The rate of change of population is proportional to its size.

4. During the process, the environment does not change in favour of one species and genetic adaptation is inconsequential.

5. Predators have limitless appetite.

As differential equations are used, the solution is deterministic and continuous. This, in turn, implies that the generations of both the predator and prey are continually overlapping.[23]

Prey

When multiplied out, the prey equation becomes

dx/dt = αx - βxy

The prey are assumed to have an unlimited food supply, and to reproduce exponentially unless subject to predation; this exponential growth is represented in the equation above by the term αx. The rate of predation upon the prey is assumed to be proportional to the rate at which the predators and the prey meet; this is represented above by βxy. If either x or y is zero then there can be no predation.

With these two terms the equation above can be interpreted as: the change in the prey's numbers is given by its own growth minus the rate at which it is preyed upon.

Predators

The predator equation becomes

dy/dt = -

In this equation, {\displaystyle \displaystyle \delta xy} represents the growth of the predator population. (Note the similarity to the predation rate; however, a different constant is used as the rate at which the predator population grows is not necessarily equal to the rate at which it consumes the prey). {\displaystyle \displaystyle \gamma y} represents the loss rate of the predators due to either natural death or emigration; it leads to an exponential decay in the absence of prey.

Hence the equation expresses the change in the predator population as growth fueled by the food supply, minus natural death.

Clone of Prey&Predator

Eleazar Puente

This diagram shows the impact of government policy and funding on a student's access to post-secondary education and the institution's health and wellness services.

P1: Systemigram

Chieng Luphuyong

Looking at the factors that influence student learning

Clone of Clone of Student Learning

Matthew Simpson

Physical meaning of the equations

The Lotka–Volterra model makes a number of assumptions about the environment and evolution of the predator and prey populations:

1. The prey population finds ample food at all times.

2. The food supply of the predator population depends entirely on the size of the prey population.

3. The rate of change of population is proportional to its size.

4. During the process, the environment does not change in favour of one species and genetic adaptation is inconsequential.

5. Predators have limitless appetite.

As differential equations are used, the solution is deterministic and continuous. This, in turn, implies that the generations of both the predator and prey are continually overlapping.[23]

Prey

When multiplied out, the prey equation becomes

dx/dt = αx - βxy

The prey are assumed to have an unlimited food supply, and to reproduce exponentially unless subject to predation; this exponential growth is represented in the equation above by the term αx. The rate of predation upon the prey is assumed to be proportional to the rate at which the predators and the prey meet; this is represented above by βxy. If either x or y is zero then there can be no predation.

With these two terms the equation above can be interpreted as: the change in the prey's numbers is given by its own growth minus the rate at which it is preyed upon.

Predators

The predator equation becomes

dy/dt = -

In this equation, {\displaystyle \displaystyle \delta xy} represents the growth of the predator population. (Note the similarity to the predation rate; however, a different constant is used as the rate at which the predator population grows is not necessarily equal to the rate at which it consumes the prey). {\displaystyle \displaystyle \gamma y} represents the loss rate of the predators due to either natural death or emigration; it leads to an exponential decay in the absence of prey.

Hence the equation expresses the change in the predator population as growth fueled by the food supply, minus natural death.

Clone of Prey&Predator

reno marcello m

This paints a broad picture for my non-profit of how tutoring helps disadvantaged youth and, with the right jump-start from a caring individual (R1 point), how learning can get learning and skill begets further skill. I appreciate any feedback to modifications because they might shape program direction.

Future iterations will show the low skilled isolated individual gets stuck in a cycle of "no-growth." I would also like to explore the dynamics of how the learner reduces dependence on the tutor.

Clone of How tutors help the learner causal loop

Steven V. Schneider, Ph.D.

From HBR study, 2014; combined with Solow Growth Model, explains why business seeks technological solutions. Yet this is not sustainable, as there is a limit to the number of STEM-trained individuals to create technological solutions.

Clone of BridgingTheGap

Wendy Kay Moran

Ejemplo Básico de Retrasos de Material

Universidad del Cauca.

Profesor: Miguel Angel Niño Zambrano

curso: Enlace Curso en Moodle

Videos ejemplos: Enlace a la lista de videos del curso youtube

Clone of Clone of Ejemplo 5 Manejo de Retrasos de Material

CaEdd

Despite a mature field of inquiry, frustrated educational policy makers face a crisis characterized by little to no clear research-based guidance and significant budget limitations -- in the face of too often marginal or unexpectedly deleterious achievement impacts. As such, education performance has been acknowledged as a complex system and a general call in the literature for causal models has been sounded. This modeling effort represents a strident first step in the development of an evidence-based causal hypothesis: an hypothesis that captures the widely acknowledged complex interactions and multitude of cited influencing factors. This non-piecemeal, causal, reflection of extant knowledge engages a neuro-cognitive definition of students. Through capture of complex dynamics, it enables comparison of different mixes of interventions to estimate net academic achievement impact for the lifetime of a single cohort of students. Results nominally capture counter-intuitive unintended consequences: consequences that too often render policy interventions effete. Results are indexed on Hattie Effect Sizes, but rely on research identified causal mechanisms for effect propagation. Note that the net causal interactions have been effectively captured in a very scoped and/or simplified format. Relative magnitudes of impact have been roughly adjusted to Hattie Ranking Standards (calibration): a non-causal evidence source. This is a demonstration model and seeks to exemplify content that would be engaged in a full or sufficient model development effort. Budget & time constraints required significant simplifying assumptions. These assumptions mitigate both the completeness & accuracy of the outputs. Features serve to symbolize & illustrate the value and benefits of causal modeling as a performance tool.

Clone of Version 10: Hattie Calibrated Education Scenario Tool Capability Demonstration

JMantilla

WIP based on Raafat Zaini's Triple Helix and PhD Colloquium and ISDC ithink models as a starting point for health care systems science modelling growth dynamics

Clone of Clone of Startup University Model

Javier Ignacio Londoño Ochoa

Exemplo de Laço de Alimentação Negativo

Clone of Temperatura do Café

Nilo Guimaraes

The purpose of this model is to study the impact different interventions with teachers have on the level of teacher use of AVID strategies.

Clone of AVID Integration at Vista Ridge High School

Beth Dees

Despite a mature field of inquiry, frustrated educational policy makers face a crisis characterized by little to no clear research-based guidance and significant budget limitations -- in the face of too often marginal or unexpectedly deleterious achievement impacts. As such, education performance has been acknowledged as a complex system and a general call in the literature for causal models has been sounded. This modeling effort represents a strident first step in the development of an evidence-based causal hypothesis: an hypothesis that captures the widely acknowledged complex interactions and multitude of cited influencing factors. This non-piecemeal, causal, reflection of extant knowledge engages a neuro-cognitive definition of students. Through capture of complex dynamics, it enables comparison of different mixes of interventions to estimate net academic achievement impact for the lifetime of a single cohort of students. Results nominally capture counter-intuitive unintended consequences: consequences that too often render policy interventions effete. Results are indexed on Hattie Effect Sizes, but rely on research identified causal mechanisms for effect propagation. Note that the net causal interactions have been effectively captured in a very scoped and/or simplified format. Relative magnitudes of impact have been roughly adjusted to Hattie Ranking Standards (calibration): a non-causal evidence source. This is a demonstration model and seeks to exemplify content that would be engaged in a full or sufficient model development effort. Budget & time constraints required significant simplifying assumptions. These assumptions mitigate both the completeness & accuracy of the outputs. Features serve to symbolize & illustrate the value and benefits of causal modeling as a performance tool.

Clone of Version 10: Hattie Calibrated Education Scenario Tool Capability Demonstration

JMantilla

This version of the CAPABILITY DEMONSTRATION model has been further calibrated (additional calibration phases will occur as better standardized data becomes available). Note that the net causal interactions have been effectively captured in a very scoped and/or simplified format. Relative magnitudes and durations of impact remain in need of further data & adjustment (calibration). In the interests of maintaining steady progress and respecting budget & time constraints, significant simplifying assumptions have been made: assumptions that mitigate both completeness & accuracy of the outputs. This model meets the criteria for a Capability demonstration model, but should not be taken as complete or realistic in terms of specific magnitudes of effect or sufficient build out of causal dynamics. Rather, the model demonstrates the interplay of a minimum set of causal forces on a net student progress construct -- as informed and extrapolated from the non-causal research literature.

Provided further interest and funding, this basic capability model may further de-abstracted and built out to: higher provenance levels -- coupled with increased factorization, rigorous causal inclusion and improved parameterization.

Clone of Clone of Version 6B: Calibrated Student-Home-Teachers-Classroom

Robert L. Brown

This version 8B of the CAPABILITY DEMONSTRATION model. A net Benefit ROI has been added. The Compare results feature allows comparison of alternative intervention portfolios. Note that the net causal interactions have been effectively captured in a very scoped and/or simplified format. Relative magnitudes and durations of impact remain in need of further data & adjustment (calibration). In the interests of maintaining steady progress and respecting budget & time constraints, significant simplifying assumptions have been made: assumptions that mitigate both completeness & accuracy of the outputs. This model meets the criteria for a Capability demonstration model, but should not be taken as complete or realistic in terms of specific magnitudes of effect or sufficient build out of causal dynamics. Rather, the model demonstrates the interplay of a minimum set of causal forces on a net student progress construct -- as informed and extrapolated from the non-causal research literature.

Provided further interest and funding, this basic capability model may further developed and built out to: higher provenance levels -- coupled with increased factorization, rigorous causal inclusion and improved parameterization.

Clone of Version 8B: Calibrated Student-Home-Teachers-Classroom-LEA-Spending

Yolande Tra